Most patients, especially patients with stable angina pectoris after treatment, the symptoms can be eased or disappeared after the establishment of adequate collateral circulation for a long time without pain in the early attacks of angina pectoris may be worsening angina pectoris and angina decubitus Central syndrome can occur as part of another heart attack, so that they “pre-infarction angina pectoris referred”

Forecast

Most patients, especially patients with stable angina pectoris after treatment, the symptoms can be eased or disappeared after the establishment of adequate collateral circulation for a long time without pain in the early attacks of angina pectoris may be worsening angina pectoris and angina decubitus Central syndrome can occur as part of another heart attack, so that they “pre-infarction angina pectoris referred”

On the pathogenesis of angina so far inconclusive debate can be traced, presented at its beginning of this century Colbeck in more than 80 years ago, the pain caused by myocardial ischemia in the stretch-induced ventricular wall (ie mechanical hypothesis) considered that 20 years later, Lewis Local pain and myocardial release of certain substances (eg chemical hypothesis) This doctrine of the two parties discussed for decades, now seems unlikely that the mechanical hypothesis to explain, has the opposite chemical hypothesis of angina pectoris was confirmed by the experiment results show ischemic heart the blood of oxygen-induced pain caused by lactic acid substances adenosine hydrogen ion and potassium ion-stimulated plasma peptide receptors, such as the heart is primarily sympathetic nerve endings of the nerve endings in the visceral organs, the distribution is far less body receptors are mainly distributed in small coronary artery proximal occurs when myocardial ischemia and hypoxia-induced pain locally produced material on the metabolism of these receptors to stimulate nerve impulses triggered by the pain of the first 1-4 sympathetic ganglion transmission to the appropriate segment of the spinal cord to the cortex through the dissemination of visceral afferent pain caused pain, which are made from the spinal cord, even in the appropriate segment of the spinal distribution of the skin where it is reflected when angina often reflects itself in the chest pain in the region is mainly located in the heart area, or substernal and left shoulder and his left arm radiation and may explain the many different types of angina pectoris

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